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SUMMARY:The role of auxin transport in bud activation - Madeline Seale fro
 m the Sainsbury Lab
DTSTART:20121130T130000Z
DTEND:20121130T133000Z
UID:TALK40561@talks.cam.ac.uk
CONTACT:24938
DESCRIPTION:An important component of plant architecture is the degree of 
 shoot branching. This is an example of phenotypic plasticity\, whereby pla
 nts change their development in response to its environment. The outgrowth
  of branches is influenced\, for example\, by light\, nutrients and planti
 ng density as well as the developmental stage of the plant. Recent evidenc
 e indicates that bud activation is initiated by canalized auxin transport 
 from the bud to the main stem giving rise to a systemic control mechanism.
  Additionally\, the phytohormones strigolactone (SL) and cytokinin (CK) in
 hibit and promote branch outgrowth respectively and it has also been propo
 sed that these hormones act locally as second messengers within buds to re
 gulate outgrowth. SL and CK alter the level of the TCP transcription facto
 r\, BRANCHED 1 (BRC1) which is expressed in dormant buds\, leading to the 
 idea that BRC1 is an integrator of branching signals. It is not clear\, ho
 wever\, whether this is a direct response to hormone signals\, or a downst
 ream effect of bud growth. To understand the role of BRC1 in more detail\,
  I have been examining its expression levels under different bud-activatin
 g treatments as well as examining how BRC1 relates to changes in auxin tra
 nsport activity. Through investigating the role of BRC1 in conjunction wit
 h the onset of bud auxin export and actual growth\, I hope to build up a p
 icture of the timing and order of events during bud activation. This will 
 contribute to understanding whether local hormone action or auxin canaliza
 tion is more important in regulating branching. 
LOCATION:Department of Plant Sciences\, Large Lecture Theatre
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