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SUMMARY:The mechanism of action of strigolactone - Catherine Taylor
DTSTART:20131206T130000Z
DTEND:20131206T132500Z
UID:TALK48222@talks.cam.ac.uk
CONTACT:Megan Cooper
DESCRIPTION:Plants are sessile and therefore their development needs to be
  environmentally responsive. One such plastic developmental process is sho
 ot branching\, where axillary meristems are produced which may remain dorm
 ant or active to produce a branch. The phytohormone auxin has been propose
 d to mediate this aspect of development by its transport. One hypothesis i
 s that axillary meristem activity depends on the canalization of auxin tra
 nsport away from the axillary meristem and out into the main stem. Auxin t
 ransport canalization involves a feedback mechanism whereby an initial flu
 x of auxin from a source to a sink upregulates the production and polariza
 tion of its own efflux transport proteins (eg. PINs)\, establishing files 
 of cells actively transporting auxin from the source to the sink. A comput
 ational model based on this mechanism can reproduce a range of branching p
 henomena. In this model\, diverse effects of a second hormone\, strigolact
 one (SL)\, can be reproduced if SL acts to promote PIN removal from the pl
 asma membrane.  This role of strigolactone was supported by experimental r
 esults in which strigolactone treatment triggered PIN1 depletion from the 
 basal plasma membrane in xylem parenchyma cells. This removal is independe
 nt of protein synthesis but dependent on clathrin-mediated endocytosis.
LOCATION:Department of Plant Sciences\, Large Lecture Theatre
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